Evidence-Based Considerations Exploring Relations between SARS-CoV-2 Pandemic and Air Pollution: Involvement of PM2.5-Mediated Up-Regulation of the Viral Receptor ACE-2

Borro, M; Di Girolamo, P; Gentile, G; De Luca, O; Preissner, R; Marcolongo, A; Ferracuti, S; Simmaco, M

HERO ID

7312540

Reference Type

Journal Article

Year

2020

Language

English

PMID

32748812

HERO ID 7312540
In Press No
Year 2020
Title Evidence-Based Considerations Exploring Relations between SARS-CoV-2 Pandemic and Air Pollution: Involvement of PM2.5-Mediated Up-Regulation of the Viral Receptor ACE-2
Authors Borro, M; Di Girolamo, P; Gentile, G; De Luca, O; Preissner, R; Marcolongo, A; Ferracuti, S; Simmaco, M
Journal International Journal of Environmental Research and Public Health
Volume 17
Issue 15
Abstract The COVID-19/SARS-CoV-2 pandemic struck health, social and economic systems worldwide, and represents an open challenge for scientists -coping with the high inter-individual variability of COVID-19, and for policy makers -coping with the responsibility to understand environmental factors affecting its severity across different geographical areas. Air pollution has been warned of as a modifiable factor contributing to differential SARS-CoV-2 spread but the biological mechanisms underlying the phenomenon are still unknown. Air quality and COVID-19 epidemiological data from 110 Italian provinces were studied by correlation analysis, to evaluate the association between particulate matter (PM)2.5 concentrations and incidence, mortality rate and case fatality risk of COVID-19 in the period 20 February-31 March 2020. Bioinformatic analysis of the DNA sequence encoding the SARS-CoV-2 cell receptor angiotensin-converting enzyme 2 (ACE-2) was performed to identify consensus motifs for transcription factors mediating cellular response to pollutant insult. Positive correlations between PM2.5 levels and the incidence (r = 0.67, p < 0.0001), the mortality rate (r = 0.65, p < 0.0001) and the case fatality rate (r = 0.7, p < 0.0001) of COVID-19 were found. The bioinformatic analysis of the ACE-2 gene identified nine putative consensus motifs for the aryl hydrocarbon receptor (AHR). Our results confirm the supposed link between air pollution and the rate and outcome of SARS-CoV-2 infection and support the hypothesis that pollution-induced over-expression of ACE-2 on human airways may favor SARS-CoV-2 infectivity.
Doi 10.3390/ijerph17155573
Pmid 32748812
Wosid WOS:000568045600001
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English
Keyword COVID-19; SARS-CoV-2; PM2.5; angiotensin-converting enzyme 2; xenobiotic response element; aryl hydrocarbon receptor