Cytogenetic effects in human exposure to arsenic

Gonsebatt, ME; Vega, L; Salazar, AM; Montero, R; Guzmán, P; Blas, J; Del Razo, LM; García-Vargas, G; Albores, A; Cebrián, ME; Kelsh, M; Ostrosky-Wegman, P

HERO ID

627248

Reference Type

Journal Article

Year

1997

Language

English

PMID

9219560

HERO ID 627248
In Press No
Year 1997
Title Cytogenetic effects in human exposure to arsenic
Authors Gonsebatt, ME; Vega, L; Salazar, AM; Montero, R; Guzmán, P; Blas, J; Del Razo, LM; García-Vargas, G; Albores, A; Cebrián, ME; Kelsh, M; Ostrosky-Wegman, P
Journal Mutation Research
Volume 386
Issue 3
Page Numbers 219-228
Abstract The cytogenetic effects of arsenic exposure were studied among rural populations that live in the same geographical area and have similar socioeconomic status, but different degree of exposure to inorganic arsenic (As) via drinking water. A group of inhabitants of Santa Ana (408.17 micrograms/l of As in drinking water) were considered the exposed individuals and a group of inhabitants of Nazareno (29.88 micrograms/l) were considered as controls. Blood and urine samples were obtained from volunteers. Past and current exposure, health, and nutritional status as well as the presence of arsenic skin lesions were ascertained in study participants through questionnaires and physical examination. The frequencies and types of chromosomal aberrations in first-division metaphases were studied in whole blood lymphocyte cultures while the presence of micronuclei (MN) was studied in exfoliated epithelial cells obtained from the oral mucosa and from urine samples. Total arsenic (TAs) content, and the relative proportions of inorganic arsenic (IAs), and the metabolites monomethylarsonic (MMA) and dimethylarsinic (DMA) acid were determined in urine samples. Exposed individuals showed a significant increase in the frequency of chromatid and isochromatid deletions in lymphocytes and of MN in oral and urinary epithelial cells. Males were more affected than females, and a higher number of micronucleated oral cells were found among those individuals with skin lesions. The type of cytogenetic damage observed gives evidence of arsenic as a clastogenic/aneugenic carcinogen.
Doi 10.1016/S1383-5742(97)00009-4
Pmid 9219560
Wosid WOS:A1997XG03500005
Url http://linkinghub.elsevier.com/retrieve/pii/S1383574297000094
Is Certified Translation No
Dupe Override 627248
Comments |WOS:A1997XG03500005
Is Public Yes
Language Text English
Keyword <?xml version="1.0" encoding="UTF-8"?><kw>DCN-240005</kw>; <?xml version="1.0" encoding="UTF-8"?><kw>Metalloids</kw>; <?xml version="1.0" encoding="UTF-8"?><kw>Environmental exposure</kw>; <?xml version="1.0" encoding="UTF-8"?><kw>Drinking water</kw>; <?xml version="1.0" encoding="UTF-8"?><kw>Humans</kw>; <?xml version="1.0" encoding="UTF-8"?><kw>Arsenic poisoning</kw>; <?xml version="1.0" encoding="UTF-8"?><kw>Genotoxic effects</kw>; <?xml version="1.0" encoding="UTF-8"?><kw>Skin lesions</kw>; <?xml version="1.0" encoding="UTF-8"?><kw>Bladder tissue</kw>; <?xml version="1.0" encoding="UTF-8"?><kw>Chromosome disorders</kw>; <?xml version="1.0" encoding="UTF-8"?><kw>Epidemiology</kw>
Is Qa No