Taurine ameliorates arsenic-induced apoptosis in the hippocampus of mice through intrinsic pathway

Li, S; Yang, L; Zhang, Y; Zhang, C; Shao, J; Liu, X; Li, Y; Piao, F

HERO ID

4242591

Reference Type

Book/Book Chapter

Year

2017

Language

English

PMID

28849454

HERO ID 4242591
Year 2017
Title Taurine ameliorates arsenic-induced apoptosis in the hippocampus of mice through intrinsic pathway
Book Title Taurine 10
Authors Li, S; Yang, L; Zhang, Y; Zhang, C; Shao, J; Liu, X; Li, Y; Piao, F
Editor Lee, DH; Schaffer, SW; Park, E; Kim, HW
Publisher Text Springer
City Dordrecht, Netherlands
Page Numbers 183-192
Abstract Our group previously reported that arsenic (As) exposure induced apoptosis in hippocampus neurons. The aim of the present study was to clarify the protective capacity of taurine (Tau) on As-induced neuronal apoptosis and the related mechanism in mouse hippocampus. Mice were divided into: control group, Tau control group, As exposure group and Tau protective group, randomly. The apoptotic rate of mouse hippocampus was determined by TUNEL staining. The levels of Bcl-2 and Bax gene and protein were analyzed by real time RT-PCR and WB, respectively. Furthermore, cytochrome c (Cyt C) release, and the activity of caspase-8 and caspase-3 were also determined. The results showed that Tau treatment induced the decrease of TUNEL-positive cells, prohibited the disturbance of Bcl-2 and Bax expression, and inhibited Cyt C release and caspase-8 and caspase-3 activation significantly. The results indicated that Tau supplement markedly ameliorates As-induced apoptosis by mitochondria-related pathway in mouse hippocampus.
Doi 10.1007/978-94-024-1079-2_16
Pmid 28849454
Wosid WOS:000428231500016
Is Certified Translation No
Dupe Override No
Conference Location Seoul, South Korea
Series Advances in Experimental Medicine and Biology, vol. 975
Isbn 9789402410778
Conference Name 20th International Taurine Meeting
Conference Date May 23-27, 2016
Is Public Yes
Language Text English
Keyword Arsenic; Taurine; Apoptosis; Hippocampus; Intrinsic pathway