Activation of liver X receptors promotes inflammatory cytokine mRNA degradation by upregulation of tristetraprolin

Xiao, J; Chen, Q; Tang, D; Ou, W; Wang, J; Mo, Z; Tang, C; Peng, L; Wang, D

HERO ID

4239762

Reference Type

Journal Article

Year

2017

Language

English

PMID

28119310

HERO ID 4239762
In Press No
Year 2017
Title Activation of liver X receptors promotes inflammatory cytokine mRNA degradation by upregulation of tristetraprolin
Authors Xiao, J; Chen, Q; Tang, D; Ou, W; Wang, J; Mo, Z; Tang, C; Peng, L; Wang, D
Journal Acta Biochimica et Biophysica Sinica
Volume 49
Issue 3
Page Numbers 277-283
Abstract Liver X receptors (LXRs) have anti-inflammatory properties. Whether LXRs play a role in post-transcriptional control of inflammatory cytokine expression is not clear. Here, we firstly identified that the synthetic LXR agonist T0901317 promoted IL-1β, IL-6 and TNFα mRNA degradation. Moreover, T0901317 destabilized TNFα mRNA through its 3'-untranslated region. In addition, T0901317 increased the expression of tristetraprolin (TTP), while antagonizing TTP with siRNA abrogated T0901317-mediated inflammatory cytokine mRNA decay. Interestingly, T0901317 repressed LPS-induced phosphorylation of ERK1/2 and p38 mitogen-activated protein kinase (MAPK) in THP-1 macrophages. The evidence presented here confirms that LXR activation with T0901317 inhibits the phosphorylation of ERK1/2 and p38 MAPK, likely resulting in the increased expression of TTP and the decay of LPS-induce inflammatory cytokine mRNAs.
Doi 10.1093/abbs/gmw136
Pmid 28119310
Wosid WOS:000397089800010
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English
Keyword liver X receptor; cytokine; mRNA decay; tristetraprolin; mitogen-activated protein kinase