A novel frameshift deletion in the COL1A1 gene identified in a Chinese family with osteogenesis imperfecta

Fan, N; Jonas, JB; He, F; Yan, NH; Wang, Y; Liu, L; Liu, DL; Zhao, L; Pang, IH; Liu, XY

HERO ID

3539504

Reference Type

Journal Article

Year

2015

Language

English

PMID

26634493

HERO ID 3539504
In Press No
Year 2015
Title A novel frameshift deletion in the COL1A1 gene identified in a Chinese family with osteogenesis imperfecta
Authors Fan, N; Jonas, JB; He, F; Yan, NH; Wang, Y; Liu, L; Liu, DL; Zhao, L; Pang, IH; Liu, XY
Journal Genetics and Molecular Research
Volume 14
Issue 4
Page Numbers 15295-15300
Abstract Osteogenesis imperfecta (OI) is a genetically heterogeneous group of disorders, characterized by abnormal bone fragility, blue sclera, deafness, joint laxity, and soft-tissue dysplasia. The purpose of this study was to elucidate the genetic or molecular basis for OI type IA in a Chinese family. We evaluated the members of a family, in which six individuals are affected with increased bone fragility and blue sclera. Results of exome sequencing revealed a novel 1-bp deletion (c.2329delG, p.A777fs) in exon 33 of the COL1A1 gene in two affected individuals, but not in a control family member without OI. The variation co-segregated with the disease in all the OI patients but not in the unaffected family members. The mutation caused a frameshift alteration after codon 777, leading to premature termination of the COL1A1 protein. Thus, our findings identified a novel frameshift deletion c.2329delG (p.A777fs) in the COL1A1 gene, which is associated with OI type IA in a Chinese family.
Doi 10.4238/2015.November.30.5
Pmid 26634493
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English