Hepatotoxic interactions of ethanol with allyl alcohol or carbon tetrachloride in rats

Berman, E; House, DE; Allis, JW; Simmons, JE

HERO ID

32567

Reference Type

Journal Article

Year

1992

Reference is

  Locked

Language

English

PMID

1522609

HERO ID 32567
In Press No
Year 1992
Title Hepatotoxic interactions of ethanol with allyl alcohol or carbon tetrachloride in rats
Authors Berman, E; House, DE; Allis, JW; Simmons, JE
Journal Journal of Toxicology and Environmental Health
Volume 37
Issue 1
Page Numbers 161-176
Abstract The effects of ethanol (64175) on allyl-alcohol (107186) and carbon-tetrachloride (56235) hepatotoxicity were studied in rats. The purpose of the study was to assess the potential for ethanol to interact with known liver toxicants when used as a solvent in the dosing vehicle. Male Fischer-344-rats were gavaged with 0, 0.05, 0.1, 0.2, or 0.5 milliliter per kilogram (ml/kg) ethanol or 21mg/kg allyl-alcohol or 20mg/kg carbon-tetrachloride alone or in combination daily for 14 days. Body weight was monitored. The rats were killed 24 hours after the last dose and livers were removed, weighed, and assayed for cytochrome P-450 (P450) and reduced glutathione (GSH). Liver sections were examined for histopathological changes. Blood samples were collected and analyzed for serum lactate-dehydrogenase (LDH), alkaline-phosphatase (ALP), alanine-aminotransferase (ALT), and aspartate-aminotransferase (AST). Ethanol alone did not significantly affect body weight gain, hepatic P450 or GSH content, or serum ALT, AST, LDH, or ALP activity. It did not cause any histologically evident liver damage, but did cause a dose related decrease in absolute and relative liver weight. The apparent threshold for these effects was between 0.05 and 0.1ml/kg. Allyl-alcohol alone caused significant increasesin absolute and relative liver weight and hepatic GSH concentration, and it induced periportal hepatocellular vacuolar degeneration. Carbon-tetrachloride alone caused significant increases in absolute and relative liver weight and serum ALT, AST, and ALP activities. It also induced centrilobular hepatocellular degeneration and necrosis. Coadministration with ethanol did not alter the effects of allyl-alcohol or carbon-tetrachloride. No significant interaction of ethanol with allyl-alcohol was determined, but a small interactive effect on body weight occurred with 0.5ml/kg ethanol and carbon-tetrachloride. The authors conclude that subacute concurrent exposure of ethanol with carbon-tetrachloride or allyl-alcohol at ethanol concentrations typically used in dosing vehicles does not cause an interactive effect or hepatotoxicity.
Doi 10.1080/15287399209531663
Pmid 1522609
Wosid WOS:A1992JQ26200012
Url http://www.tandfonline.com/doi/pdf/10.1080/15287399209531663#.VIn_6THF9qE
Is Certified Translation No
Dupe Override No
Comments J. Toxicol. Environ. Health 37: 161-176.
Is Public Yes
Language Text English
Is Qa Yes
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