Alteration of the microRNA-122 regulatory network in rat models of hepatotoxicity

Lardizábal, MN; Rodríguez, RE; Nocito, AL; Daniele, SM; Palatnik, JF; Veggi, LM

HERO ID

2956069

Reference Type

Journal Article

Year

2014

Language

English

PMID

24388909

HERO ID 2956069
In Press No
Year 2014
Title Alteration of the microRNA-122 regulatory network in rat models of hepatotoxicity
Authors Lardizábal, MN; Rodríguez, RE; Nocito, AL; Daniele, SM; Palatnik, JF; Veggi, LM
Journal Environmental Toxicology and Pharmacology
Volume 37
Issue 1
Page Numbers 354-364
Abstract MicroRNAs are small RNA molecules that post-transcriptionally regulate gene expression. MicroRNA-122 is the most abundant and specific liver microRNA. Hepatotoxicity involves a significant alteration of liver gene expression. The aim of this work was to evaluate the microRNA-122 regulatory network in models of hepatotoxicity induced by thioacetamide or carbon tetrachloride. We report that the toxins decreased the expression of microRNA-122, which corresponded with an increase in two target genes: Cyclin G1 and the cationic amino acid transporter CAT-1. We found a decreased expression of its precursor, pri-microRNA-122, and of the transcription factors that specifically bind its promoter: CCAAT/enhancer-binding protein alpha, and members of the hepatocyte nuclear factor family. Therefore, microRNA-122 expression levels are under transcriptional control during hepatotoxicity. We propose that the changes observed are associated with the liver response to cope with the injury caused by the hepatotoxins, likely through a cell proliferation process to repair the damaged tissue.
Doi 10.1016/j.etap.2013.12.008
Pmid 24388909
Wosid WOS:000332919800039
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English