Environmental chemical exposures and autism spectrum disorders: A review of the epidemiological evidence

Kalkbrenner, AE; Schmidt, RJ; Penlesky, AC

HERO ID

2535290

Reference Type

Journal Article

Subtype

Review

Year

2014

Language

English

PMID

25199954

HERO ID 2535290
Material Type Review
In Press No
Year 2014
Title Environmental chemical exposures and autism spectrum disorders: A review of the epidemiological evidence
Authors Kalkbrenner, AE; Schmidt, RJ; Penlesky, AC
Journal Current Problems in Pediatric and Adolescent Health Care
Volume 44
Issue 10
Page Numbers 277-318
Abstract In the past decade, the number of epidemiological publications addressing environmental chemical exposures and autism has grown tremendously. These studies are important because it is now understood that environmental factors play a larger role in causing autism than previously thought and because they address modifiable risk factors that may open up avenues for the primary prevention of the disability associated with autism. In this review, we covered studies of autism and estimates of exposure to tobacco, air pollutants, volatile organic compounds and solvents, metals (from air, occupation, diet, dental amalgams, and thimerosal-containing vaccines), pesticides, and organic endocrine-disrupting compounds such as flame retardants, non-stick chemicals, phthalates, and bisphenol A. We included studies that had individual-level data on autism, exposure measures pertaining to pregnancy or the 1st year of life, valid comparison groups, control for confounders, and adequate sample sizes. Despite the inherent error in the measurement of many of these environmental exposures, which is likely to attenuate observed associations, some environmental exposures showed associations with autism, especially traffic-related air pollutants, some metals, and several pesticides, with suggestive trends for some volatile organic compounds (e.g., methylene chloride, trichloroethylene, and styrene) and phthalates. Whether any of these play a causal role requires further study. Given the limited scope of these publications, other environmental chemicals cannot be ruled out, but have not yet been adequately studied. Future research that addresses these and additional environmental chemicals, including their most common routes of exposures, with accurate exposure measurement pertaining to several developmental windows, is essential to guide efforts for the prevention of the neurodevelopmental damage that manifests in autism symptoms.
Doi 10.1016/j.cppeds.2014.06.001
Pmid 25199954
Wosid WOS:000344821300002
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English