Perfluorooctane sulfonamide-mediated modulation of hepatocellular lipid homeostasis and oxidative stress responses in Atlantic Salmon Hepatocytes

Wågbø, A; Cangialosi, MV; Cicero, N; Letcher, RJ; Arukwe, A

HERO ID

2187308

Reference Type

Journal Article

Year

2012

Language

English

PMID

22594583

HERO ID 2187308
In Press No
Year 2012
Title Perfluorooctane sulfonamide-mediated modulation of hepatocellular lipid homeostasis and oxidative stress responses in Atlantic Salmon Hepatocytes
Authors Wågbø, A; Cangialosi, MV; Cicero, N; Letcher, RJ; Arukwe, A
Journal Chemical Research in Toxicology
Volume 25
Issue 6
Page Numbers 1253-1264
Abstract We have investigated the effects of perfluorooctane sulfonamide (PFOSA) on cellular functions and lipid homeostasis (including beta-oxidation) in salmon primary hepatocytes. Salmon hepatocytes were exposed to PFOSA at 0 (control), 2, 20, and 50 mu M for 12 and 24 h. Fatty acids (FM) and lipids were determined by GC-MS; FA elongase (FAE), Delta 5-desaturase (FADS), Delta 6-desaturase (FAD6), peroxisome proliferator-activated receptors (PPARs), acyl coenzyme A (ACOX-1), glutathione peroxidase (GPx), catalase (CAT), and glutathione S-transferase (GST) mRNA were analyzed using qPCR. GST activity was analyzed by biochemical assays using 1-chloro-2,4-dinitrobenzene (CDNB) as substrate. Our data showed that PFOSA produced significant changes in FA composition that predominantly involved a decrease (at 12 h) and an increase (at 24 h) in FA methyl esters (FAMEs), MUFA, total PUFA, and (n-3 and n-6) PUFA. Particularly, an increase of alpha-linolenic acid (ALA; 18:3n-3), eicosapentaenoic acid [EPA; 20:5n-3], and arachidonic acid [ARA: 20:4n-6] with associated increase in FAE, FADS, and FAD6 mRNA were observed after PFOSA exposure, while cis-13,16-docosadienoic acid (22:2) was significantly decreased. PFOSA produced apparent concentration-dependent increase of PPAR alpha and PPAR gamma. CAT, GPx, and GST mRNA show that PFOSA produced concentration- and time-specific increase of CAT and GST, but no changes in GST activity were observed. In general, these responses indicate that PFOSA evokes deleterious effects on cellular lipid homeostasis a;ad transcriptional responses that regulate cellular oxidative homeostasis in salmon hepatocytes.
Doi 10.1021/tx300110u
Pmid 22594583
Wosid WOS:000305300100011
Is Certified Translation No
Dupe Override No
Comments Scopus URL: https://www.scopus.com/inward/record.uri?eid=2-s2.0-84862518194&doi=10.1021%2ftx300110u&partnerID=40&md5=6734c04339d31640e2d006d053fb0d72
Is Public Yes
Language Text English