Cardiovascular system and kidney as specific targets of chronic exposure to vanadate in the rat: Functional and morphological findings

Carmignani, M; Boscolo, P; Volpe, AR; Togna, G; Masciocco, L; Preziosi, P

HERO ID

1466080

Reference Type

Book/Book Chapter

Year

1991

Language

English

PMID

1805718

HERO ID 1466080
Year 1991
Title Cardiovascular system and kidney as specific targets of chronic exposure to vanadate in the rat: Functional and morphological findings
Book Title Recent Developments in Toxicology: Trends, Methods and Problems
Authors Carmignani, M; Boscolo, P; Volpe, AR; Togna, G; Masciocco, L; Preziosi, P
Editor Chambers, PL; Chambers, CM; Wiezorek, WD; Golbs, S
Publisher Text Springer
City Heidelberg, Germany
Volume 14
Page Numbers 124-127
Abstract The physiological significance of vanadium (Va) and its mechanisms of cardiovascular (CV) and renal toxicity (following chronic exposure) remain contradictory (Nechay 1984). Pentavalent vanadate (Va+5, as analogue of phosphate) and tetravalent vanadyl (resembling the size of Mg+2) have many potential sites of action. However, it is yet to be established whether CV and renal effects of Va involve inhibition and/or stimulation of various enzymatic activities (for instance, Na+, K+, ATPase and adenylate cyclase, respectively) and are influenced by other ions (like Na+, K+, Mg+2 and Mn+2) and/or by the redox and metabolic states of the involved cells. Contradictory results also concern the possibility that Va is implicated in certain types of arterial hypertension (Carmignani et al 1990).
Doi 10.1007/978-3-642-74936-0_24
Pmid 1805718
Is Certified Translation No
Dupe Override No
Conference Location Leipzig, Germany
Series Archives of Toxicology
Isbn 9783642749360
Conference Name European Societies of Toxicology Meeting
Conference Date September 12–14, 1990
Is Public Yes
Language Text English
Keyword Instrumental Neutron Activation Analysis Chronic Exposure Proximal Tubule Cell Cardiac Inotropism Pentavalent Vanadate