Dietary sodium gluconate protects rats from large bowel cancer by stimulating butyrate production

Kameue, C; Tsukahara, T; Yamada, K; Koyama, H; Iwasaki, Y; Nakayama, K; Ushida, K

HERO ID

1457148

Reference Type

Journal Article

Year

2004

Language

English

PMID

15051851

HERO ID 1457148
In Press No
Year 2004
Title Dietary sodium gluconate protects rats from large bowel cancer by stimulating butyrate production
Authors Kameue, C; Tsukahara, T; Yamada, K; Koyama, H; Iwasaki, Y; Nakayama, K; Ushida, K
Journal Journal of Nutrition
Volume 134
Issue 4
Page Numbers 940-944
Abstract Butyrate has an antitumorigenic effect on colorectal cancer cell lines. Dietary sodium gluconate (GNA) promotes butyrate production in the large intestine. Accordingly, we examined the effect of dietary GNA on tumorigenesis in the large intestine in rats. Male Fisher-344 rats (n = 32) were divided into 4 groups: 2 diets (with or without 50 g GNA/kg basal diet) X 2 treatments (with or without carcinogen administration). Colonic tumors were induced by 3 intraperitoneal injections of azoxymethane (115 mg/kg body wt, 1 time/wk) and dietary deoxycholic acid (2 g/kg basal diet). The experiment was conducted for 33 wk except for a few rats. Ingestion of GNA increased cecal butyrate concentration at the end of experiment (P < 0.01). No tumor development occurred in the untreated groups. Ingestion of GNA decreased the incidence of tumors in rats administered the carcinogen (37.5 vs. 100%, P < 0.05). Ingestion of GNA also decreased the mean number of tumors per rat (0.5 +/- 0.8 vs. 2.8 +/- 1.5, P < 0.01). beta-Catenin accumulation and TdT-mediated dUTP nick end labeling (TUNEL) positive cells in tumors were histochemically examined. The results of this study suggested that the antitumorigenic effect of GNA may involve the stimulation of apoptosis through enhanced butyrate production in the large intestine.
Doi 10.1093/jn/134.4.940
Pmid 15051851
Wosid WOS:000220681700038
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English
Keyword sodium gluconate; butyrate; colorectal cancer prevention; apoptosis; rat
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